The NCAA has gone too far if an athlete de facto needs dexamethasone sodium phosphate in order to prevent cerebral hypoxia and survive to the end of a race, session or season. Correspondingly, it is noteworthy to understand that pharmacological methodologies for achieving metabolic anabolism, during the requisite sustained exercise, have no efficacy relative to any congenital impedance of cranial tissue blood flow. Thus, the psychiatric question becomes “can capillary hypoxic damage be diagnosed as mood or personality disorder or a pertaining comorbidity”. In other words; does metabolic anabolism induction pharmacotherapy disproportionately neglect the brain as muscle catabolism is eliminated and oxygen delivery elsewhere is sustained?